Tìm thấy 20+ kết quả cho từ khóa "Lung injury"
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Comparison of two fluid-management strategies in acute lung injury. Effects of sevoflurane preconditioning on lung injury during one lung ventilation
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Cytokines in tolerance to hyperoxia-induced injury in the developing and adult lung. Manganese superoxide dismutase gene-modified mesenchymal stem cells attenuate acute radiation-induced lung injury. Heme oxygenase-1 in lung disease. Induction of heme oxygenase-1 by hemin protects lung against orthotopic autologous liver transplantation-induced acute lung injury in rats. Desoxyrhapontigenin up-regulates Nrf2- mediated heme oxygenase-1 expression in macrophages and inflammatory lung injury.
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Hydrogen inhalation therapy has been proven effective in mitigating in several animal models of lung injury includ- ing hyperoxic lung injury, hemorrhagic shock-induced lung injury, radiation-induced lung injury, and bronchial asthma .
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Oxidative lung injury correlates with one-lung ventilation time during pulmonary lobectomy: a study of exhaled breath condensate and blood. Effects of OLV preconditioning and postconditioning on lung injury in thoracotomy
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Paravertebral dexmedetomidine as an adjuvant to ropivacaine protects against independent lung injury during one-lung ventilation: a preliminary randomized. Background: To investigate the effect of paravertebral dexmedetomidine as an adjuvant to ropivacaine on independent lung injury during one-lung ventilation..
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Animal models of acute lung injury. ROS Signaling in the Pathogenesis of Acute Lung Injury (ALI) and Acute Res- piratory Distress Syndrome (ARDS). The adult respiratory distress syndrome cognitive outcomes study: long-term neuropsycho- logical function in survivors of acute lung injury. Neurological outcome after experimental lung injury. Long-term simvastatin attenuates lung injury and oxidative stress in murine acute lung injury models induced by oleic Acid and endotoxin.
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H&E staining results showed that miR-26a-5p upregula- tion alleviated the OIP5-AS1 overexpression caused lung injury in ALI mice (Fig. 3 MiR-26a-5p inhibited the progression of lung injury by regulating TLR4. 0.05 compared with ALI + AAV-miR-26a-5p + AAV-vector group in C–F. 0.05 compared with LPS + miR-26a-5p + vector group in G, H. was increased after OIP5-AS1 upregulation, while miR- 26a-5p overexpression rescued the effect of OIP5-AS1 upregulation (Fig.
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Lung injury score by both histological and morphometric analysis. Compared with group NC, the he lung injury score was markedly increased in group HV (p = 0.000). In group HV + DFO, the lung injury score (p = 0.047) was increased to.
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In the present study, lung tissue pathology and the in- crease of lung tissue W/D ratio suggested that mechan- ical ventilation with hyperoxia for 4 h caused lung injury..
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5p, thereby reducing the inflammatory injury of lung cells [9].
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Effects of intraoperative lung-protective ventilation on clinical outcomes in patients with traumatic brain injury: a randomized controlled trial. Background: Secondary lung injury is the most common non-neurological complication after traumatic brain injury (TBI). This study aimed to investigate whether intraoperative LPV could improve respiratory function and prevent postoperative complications in emergency TBI patients..
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Protective effect of Dexmedetomidine on endotoxin-induced acute lung injury in rats. Recruitment of lung volume during surgery neither affects the postoperative spirometry nor the risk of hypoxaemia after laparoscopic gastric bypass in morbidly obese patients: a randomized controlled study
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Alveolar hypoxia induces inflam- mation and the use of AOI to maintain a certain swelling of the lung can delay the occurrence of lung injury [15].
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Mechanical ventilation with lower tidal volumes and positive end-expiratory pressure prevents alveolar coagulation in patients without lung injury.. Wolthuis EK, Choi G, Dessing MC, Bresser P, Lutter R, Dzoljic M, et al.. Mechanical ventilation with lower tidal volumes and positive end-expiratory pressure prevents pulmonary inflammation in patients without preexisting lung injury. Jaber S, Coisel Y, Chanques G, Futier E, Constantin JM, Michelet P, et al.
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In one-lung ventilation (OLV), it is indicated that high V T and inspiratory airway pressure are risk factors for acute lung injury after thoracic surgery [9–11], while high ventilator support is sometimes needed during OLV to maintain patient’s oxygenation and eliminate carbon dioxide. However, the evidence for optimal venti- lator settings during OLV remains insufficient.
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A number of human and animal studies have implicated the neutrophil as one of the key cellular mediators of this early phase of acute lung injury.
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Ventilator-induced lung injury during con- trolled ventilation in patients with acute respiratory distress syndrome:. Silva PL, Ball L, Rocco PA, et al. Power to mechanical power to minimize ventilator-induced lung injury? Intensive Care Med Exp. Mechanisms of ventilator-induced lung injury in healthy lungs. Ventilator-induced lung injury. Gattinoni L, Carlesso E, Cadringher P, et al. Gattinoni L, Tonetti T, Cressoni M, et al. Ventilator-related causes of lung injury: the mechanical power.
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Electrical impedance tomography compared to positron emission tomography for the measurement of regional lung ventilation: an experimental study. Assessment of regional ventilation distribution: comparison of vibration response imaging (VRI) with electrical impedance tomography (EIT). Effect of positive end-expiratory-pressure on regional ventilation in patients with acute lung injury evaluated by electrical impedance tomography.
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Reduced local immune response with continuous positive airway pressure during one-lung ventilation for oesophagectomy. The effect of positive endexpiratory pressure and continuous positive airway pressure on the oxygenation and shunt fraction during one-lung ventilation with propofol anesthesia. Lung injury after one-lung ventilation: a review of the pathophysiologic mechanisms affecting the ventilated and the collapsed lung. https://doi.org/10.1213/ANE..
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Increasing I:E from 1:2 to 1:1 and 2:1 led to significant elevations in mean airway pressure regardless of the presence of injury (p <. circles: mean airway pressure. Figure 2 summarizes the effects of lung injury and the changes in I:E ratio on brain and peripheral muscle tissue oxygenation as measured by NIRS. Induction of lung injury resulted in significant deterioration in these indices at the lower PEEP level (p <.