Tìm thấy 20+ kết quả cho từ khóa "Cerebral blood flow"
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The methodological limitations are that trans- cranial Doppler does not measure cerebral blood flow, only changes of the blood flow velocities are propor- tional to cerebral blood flow values. And finally, in the present study we only tested CO 2 -reactivity toward hypocapnic direction.
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Cerebral autoregulation is the inherent ability of the brain circulation to maintain constant cerebral blood flow over wide ranges of systemic blood pressure [17].. cerebral blood flow ¼ cerebral perfusion pressure cerebrovascular resistance. Assuming that cerebral perfusion pressure is propor- tional to the mean arterial pressure, if MAP decreases, a vasodilation of the resistance arterioles should occur in order to maintain a constant cerebral blood flow.
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Transcranial measurement of blood velocities in the basal cerebral arteries using pulsed Doppler ultrasound: velocity as an index of flow. Cerebral blood flow velocity in relation to cerebral blood flow, cerebral metabolic rate for oxygen, and electroencephalogram analysis during isoflurane anesthesia in dogs. Effect of haematocrit on cerebral blood-flow in man.. The effect of hematocrit on cerebral blood flow velocity in neonates and infants undergoing deep hypothermic cardiopulmonary bypass.
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In steady state, cerebral blood flow is maintained constant with static cerebral autoregulation [4]. In acute change in blood pressure, cerebral blood flow is com- pensatory adjusted by dynamic cerebral autoregulation [12, 13]. If blood pressure increased suddenly, cerebral blood flow may increase transiently. After the Trendelenburg position combined with CO 2.
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As the semiquantitative variables of the O2C system are not validated by invasive measurements of cerebral blood flow in this study, the rela- tive increase in local cerebral blood flow cannot be depicted in absolute values. Therefore, a direct implica- tion for a significant reduction of DCI or cerebral vaso- spasm cannot be drawn due to the small sample size and lack of randomization..
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Influence of PEEP on cerebral blood flow and cerebrovascular autoregulation in patients with acute respiratory distress syndrome.. Suzuki S, Takasaki S, Ozaki T, et al. The physiologic effects of inverse ratio ventilation. Inverse ratio ventilation (I/E = 2/1) in acute respiratory distress syndrome: a six-hour controlled study. Effects of inverse ratio ventilation and positive end- expiratory pressure in oleic acid-induced lung injury.
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The OR value of dHB indi- cates that when increasing amounts of hemoglobin are lost cerebral oxygen saturation reduction are at increases risk (Table 3). A previous study found that if brain metab- olism is constant, cerebral oxygen saturation directly re- flects cerebral blood flow [5]. Cerebral oxygen saturation monitoring has therefore been widely used in adult and pediatric surgery [6, 7].
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Cortical blood flow and cerebral perfusion pressure in a new noncraniotomy model of subarachnoid hemorrhage in the rat. Magnesium and the inflammatory response: potential pathophysiological implications in the management of patients with aneurysmal subarachnoid hemorrhage?. Local distribution of the effects of sympathetic stimulation on cerebral blood flow in the rat. Neurogenic Mechanisms in the Cerebrovascular Bed..
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Prog ressive decrease in pulse and cardiac output;. decrease in carbon dioxide production per 8°C drop in temperature. increase in renal blood flow;. renal autoreg ulation intact;. L oss of cerebro vascula r autoreg ulation;. decline in cerebral blood flow;. Prog ressive decrease in blood. pressure, heart rate, and cardiac output. decrease in oxygen consumptio n. D ecrease in renal blood flow parallels decreas e in cardiac output;. progres sive decreas e in EEG. decreas e in basal metabol ism
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Using MRI, we showed that at the age of 12 months, when AD was progressing, OXYS rats had structural and functional alterations in the cerebral blood flow typical of chronic ischemia such as reduced cerebral blood flow and a decline of cerebrovascular reactivity in response to a vaso- dilatory challenge, also defined as the cerebrovascular re- serve [11, 12], as in patients with AD.
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The effects of propofol on intracranial pressure and cerebral perfusion pressure in patients with brain tumors. Sedation by constant-rate propofol in the head-injured patient. Propofol versus isoflurane anesthesia under hypothermic conditions: effects on intracranial pressure and local cerebral blood flow after diffuse traumatic brain injury in the rat.
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However, in a retrospective analysis in children younger than 6 months conducted by Rhondali et al., the authors com- pared data of two studies investigating the impact of sevo- flurane anesthesia on cerebral blood flow by transcranial. Even more Rhon- dali et al. 2 Time course of the median sevoflurane concentration (median ± interquartile range) calculated from patients in the group of combined general- and caudal anesthesia.
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Roher and partners point out the AD group’s cerebral blood flow is 20% lower than that in the healthy elderly group and these values are directly correlated with pulse pressure and cognitive tests.. MTA scores: In our research, the MTA score only increases in the right brain by age group with p <.
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Although the difference was not statistically significant, there was no monitoring of cerebral blood flow during the operation, and the effect of methoxa- mine on cerebral blood flow in patients should be fur- ther explored in future studies. Finally, although the mode of operation in this study included endoscopy and laparotomy, the data of the two groups were evenly distributed.
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Reduced cerebral blood flow as a consequence of a rapid decline in Pa CO2 may cause dizziness, mental confusion, and seizures, even in the absence of hypoxemia
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This will ensure appropriate therapy and may prevent delivery of inappropriate therapy (chest compressions associated with cardiopulmonary resuscitation) that may inflict trauma.Patients who have lost consciousness should be placed in a position that maximizes cerebral blood flow, offers protection from trauma, and secures the airway. Whenever possible, the patient should be placed supine with the head turned to the side to prevent aspiration and the tongue from blocking the airway.
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Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. A report of the American College of Cardiology/American Heart Association task force on practice guidelines (committee on Management of Acute Myocardial Infarction). In-vivo videomicroscopy of mesenteric postcapillary venules after cardiac arrest in rats: A-200. Role of inflammation after cardiac arrest.
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Li M, Kuroiwa Y, Wang L, Kamitani T, Omoto S, Hayashi E, Takahashi T, Suzuki Y, Koyano S, Ikegami T, Matsubara S (2005) Visual event-related potentials under different interstimulus intervals in Parkinson’s disease: relation to motor disability, WAIS-R, and regional cerebral blood flow. Li M, Kuroiwa Y, Wang L, Kamitani T, Takahashi T, Suzuki Y, Omoto S (2003) Early sensory information processes are enhanced on visual oddball and S1–S2 tasks in Parkinson’s disease:.
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Though maintenance of cerebral perfusion pressure (CPP) may not be as critical in these children due to lack of cerebral structures, an agent that de- creases ICP and maintains hemodynamics is still benefi- cial to avoid seizures, aspiration, and maintain cardiac output to other vital organs.. Although keta- mine is a relative contraindication with increased ICP due to its ability to increase cerebral metabolic rate, it does preserve cerebral blood flow and mean arterial pressure.
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The rapid cessation of action is largely due to redistribution: after intravenous injection, brain concentra- tion climbs rapidly to anesthetic levels because of the high cerebral blood flow;. the drug then distributes evenly in the body, i.e., concentration rises in the pe- riphery, but falls in the brain—redistri- bution and cessation of anesthesia (A)..