Tìm thấy 14+ kết quả cho từ khóa "Inflammatory markers"
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RESEARCH ON CHANGES IN LEVELS OF NON-SPECIFIC INFLAMMATORY MARKERS IN PATIENTS WITH ACUTE. Objectives: To investigate serum levels of some inflammatory markers (CRP-hs, C3, C4, and Interleukin (IL)-6 in patients with acute coronary syndrome (ACS).
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FREE FATTY ACID ENHANCES EXPRESSION OF INFLAMMATORY MARKERS IL6, TLR2, TLR4 IN C2C12 MUSCLE CELLS. In the present study, we differentiate C2C12 myocytes into mature myotubes. Then the cells were treated with free fatty acid (FFA) to mimic the obese microenvironment.The result showed that the level of IL6 mRNA, a key inflammatory cytokine, was significantly higher in the myotubes treated with FFA compared to the myotubes treated with the control medium..
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Preemptive oxycodone is superior to equal dose of sufentanil to reduce visceral pain and inflammatory markers after surgery: a randomized controlled trail. Background: Postoperative visceral pain is common after surgery and previous studies have demonstrated that oxycodone is an effective treatment.
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Routine clinical markers of the magnitude of the systemic inflammatory response after elective operation:. Crystalloid or colloid for goal- directed fluid therapy in colorectal surgery. The endothelial glycocalyx: a review of the vascular barrier
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Cardiac surgery is frequently accompanied by non-infective systemic inflammatory response syndrome (SIRS) induced by surgical trauma, CPB, organ ischaemia-reperfusion in- juries, a change in the body temperature and endotoxin release [33, 34]. risk of mortality in the postoperative period . PCT is one of the accepted and widely used inflammatory markers, and it is also applied for follow-up in the cardiac surgery setting [24, 25].
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Markers of the SIR syndrome (SIRS), in- cluding CRP and PCT, as well as white blood cell count (WBC) have been shown to be prognostic of survival in patients in a variety of cancers [8–13]. We therefore conducted a mul- ticenter, prospective, observational study to examine the possible independent relationships between the blood concentrations of the abovementioned inflammatory markers at ICU admission and ICU mortality in critically ill adults.
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One lung ventilation resulted in an increase in the measured inflammatory markers in both groups (cal- culated as difference in concentrations of inflamma- tory markers in serum, performed after OLV at T 3. however, the increase of in- flammatory markers upon OLV in the group C was significantly higher than those of group T. Meanwhile, the inflammatory markers of group C was signifi- cantly higher than those of group T in BALF at T 3 .
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Third, in the present study, we only focused on the effects of different analgesic modalities on systemic inflammatory response but not local inflammatory response at the site of tissue injury. Fourth, because the impact of different analgesic modalities on in- flammatory response to craniotomy has been rarely re- ported, in the current pilot study, the sample size calculation was done based on MAP and not inflammatory markers in response to craniotomy.
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When inflammatory markers that predicted PFS were also included in the multivari- able model, better PFS was predicted by an LMR of ≥5 among patients who received capecitabine monotherapy (p=0.03) and a PLR of <250 among patients who received eribulin monotherapy (p=0.005) (Table 2)..
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Initiate results showed that, after treatment, the concentration of inflammatory markers reduced significantly, especially IL-6 reduced from 16.59 pg/ml to 3.99 pg/ml, and hs-CRP reduced from 8.7 mg/l to 1.3 mg/l. Conclusion: The concentration of non-specific inflammatory markers IL-6 and hs-CRP decreased post-treatment suggesting those as biomarker for monitoring progress and prognosis of patients with ACS..
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It has also been shown to reduce apoptosis and improve spermatogenesis in rats with varicocele (Mazhari et al., 2018) and to have ameliorating effects on lipid peroxidation and inflammatory processes in lung ischemia/reperfusion injury (Jin et al., 2016).. Torsion 3 h / detorsion 3 h group. In the study, 720° torsion was applied to the left testicle.
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Although cisplatin exerted a definite oxidative stress on the kidneys in our experimental model, renal tissue did not exhibit any statistically significant change in the inflammatory markers except IFN-γ. Based on these findings, it could be considered that the inflammation-related toxic effect of cisplatin on kidneys occurs through IFN-γ, as previously observed in the study by Kimura et al.
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However, previous studies mainly addressed the effects of anesthetics on release of inflammatory markers without showing enough data on postoperative clinical outcomes. To our knowledge, few studies have systematically compared the effects of propofol and sevoflurane on the development of PP after esophageal surgery. In our study, we found that two different anesthesia methods had similar effects on the PP rate in patients undergoing esophagectomy.
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Increased serum inflammatory markers in the absence of clinical and skeletal muscle inflammation in patients with chronic obstructive pulmonary disease. Interferon gamma induction of pulmonary emphysema in the adult murine lung. Arginase: an emerging key player in the mammalian immune system. ness of the Medical Research Council (MRC) dyspnoea scale as a measure of disability in patients with chronic obstructive pulmonary disease. Development and first validation of the COPD Assessment Test.
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Background: Ionizing Radiation (IR) is a known pro-inflammatory agent and in the process of development of biomarkers for radiation biodosimetry, a chronic inflammatory disease condition could act as a confounding factor.. with that of a normal wild-type mouse to potentially develop transcriptomics-based biodosimetry markers that can predict radiation exposure in individuals regardless of pre-existing inflammatory condition.. 10%) after radiation exposure in Il10.
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Inflammation is recognised as one of the hallmarks of cancer as the tumor-associated inflammatory response in the tumour microenvironment leads to tumorigen- esis and progression [5].
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Surgical stress and anesthesia induce inflammatory responses by disturbing the balance between pro- and anti- inflammatory cyto- kines [8], which may result in aggravation of the neu- roinflammatory response in PD patients. We chose to use sevoflurane, one of the commonly used inhalational anesthetics, because it helps rapid induction and recovery. Shan et al.
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Cuprizone treatment induced expression of various inflammatory cells and cytokines as immune response Eight weeks of cuprizone treatment leading to demyelination triggered alterations in the level of various pro- and anti-inflammatory cytokines at different times, indicating a hypothetical sequence of immune cell recruitment and activation. IFN-gamma level increased to at the 6th and 8th week of cuprizone treatment and decreased to over 16 weeks (p <.
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To determine the effect of sevoflurane on neuroin- flammation, expression levels of the inflammatory. markers IL-6, IL-8 and TNF-α were measured in the cortex and hippocampus of rats in the control, L-Sev and H-Sev groups by Western blotting. IL-6, IL-8 and TNF-α expression increased significantly in the cortex and hippocampus after low or high-dose sevoflurane anesthesia compared with that of the control group (Fig.
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These markers are viewed as speaker-oriented markers, which emphasize the subjective attitude of the speaker towards the message. These markers are said to attenuate the speaker meaning by increasing the degree of subjectivity of the utterance. The use of Evidential modal markers of low certainty in the speaker’s assertion can function as hedging devices which are downgraders. That may lead the speaker to the situation of doubt and suspicion.