Tìm thấy 12+ kết quả cho từ khóa "Pulmonary arterial hypertension"
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Evaluation and classification of pulmonary arterial hypertension.. Borderline mean arterial pressure in patients with systemic sclerosis: transpulmonary gradient predicts risk of developing pulmonary hypertension. Evidence based pulmonary arterial hypertension in systemic sclerosis:. Early 408 detection of pulmonary arterial hypertension in systemic sclerosis:. Compari- son of the predictive value of three screening models for pulmonary arte- rial hypertension in systemic sclerosis.
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Early results of mitral valve replacement in severe pulmonary artery hypertension - An institutional prospective study. Mitral valve replacement in severe pulmonary arterial hypertension
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Haemo- dynamic phenotypes and survival in patients with systemic sclerosis: the impact of the new definition of pulmonary arterial hypertension. Clinical characterization and survival of patients with borderline elevation in pulmonary artery pressure. definition of pulmonary hypertension on the prevalence of pre-capillary pulmonary hypertension. Significance of pulmonary arterial pressure and diffusion capacity of the lung as prognosticator in patients with idiopathic pulmonary fibrosis..
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Treatment of right ventricular dysfunction and heart failure in pulmonary arterial hypertension. Severe pulmonary hypertension and right ventricu‑. Ryan JJ, Thenappan T, Luo N, et al. Vachiéry JL, Adir Y, Barberà JA, et al. Pulmonary hypertension due to left heart diseases. How to define pulmonary hypertension due to left heart disease. Nobuyoshi M, Arita T, Shirai S, et al. Fang JC, DeMarco T, Givertz MM, et al.
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Effect of atorvastatin on pulmonary arterial hypertension in rats through PI3K/AKT signaling pathway. bFGF overexpression adipose derived mesenchymal stem cells improved the survival of pulmonary arterial endothelial cells via PI3k/Akt signaling pathway. PDGF promotes the warburg effect in pulmonary arterial smooth muscle cells via activation of the PI3K/AKT/mTOR/HIF-1alpha signaling pathway.
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Combined pulmonary fibrosis and emphysema: a distinct underrecognised entity. Annual changes in pulmonary function in combined pulmonary fibrosis and emphysema: over a 5-year follow-up. Combined pulmonary fibrosis and emphysema alters physiology but has similar mortality to pulmonary fibrosis without emphysema. Idiopathic pulmonary fibrosis and emphysema: decreased survival associated with severe pulmonary arterial hypertension.
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Tăng áp động mạch phổi ( pulmonary arterial hypertension – PAH) được định nghĩakhi mPAP ≥ 25 mmHg, áp lực động mạch phổi bít ( PAWP. Yếu tố khởi phátNhững nguyên nhân có thể làm xấu đi tình trạng lâm sàng của bệnh nhân PAH gồmnhiễm trùng phổi, ho ra máu, rối loạn nhịp nhanh trên thất, thiếu máu, cường hoặc suygiáp, phù, suy thận, gắng sức quá mức, giảm liều lợi tiểu, hoặc ngưng thuốc điều trị dãnmạch phổi đặc. KMĐM: khí máu động mạch. PAWP: áp lực động mạch phổibít.
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Tăng huyết áp phổi (pulmonary hypertension) là một bệnh có dự hậu xấu do nhiều nguyên nhân khác nhau. Sự điều trị nhằm vào loại trừ nguyên nhân và trong một số trường hợp dùng các chất giãn mạch. Cần làm nhiều xét nghiệm khác nhau để tìm các nguyên nhân làm tăng huyết áp, chỉ sau khi đã tìm mà không thấy nguyên nhân mới chấp nhận chẩn đoán tăng huyết áp động mạch phổi vô căn (idiopathic pulmonary arterial hypertension).
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Mitral valve replacement in severe pulmonary hypertension.. following mitral valve replacement in man.. Early reduction of pulmonary vascular resistence after mitral valve replacement.. The early course of pulmonary hypertension in patients undergoing mitral valve replacement with cardioplegic arrest. Mitral valve replacement in severe pulmonary arterial hypertension
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Endothelial dysfunction in pulmonary arterial hypertension: an evolving landscape (2017 Grover Conference Series). Endothelial dysfunction in the pulmonary vascular bed.. Flow mediated dilation of the brachial artery: an investigation of methods requiring further standardization. Thijssen D, Black MA, Pyke KE, Padilla J, Atkinson G, Harris RA, et al.. Assessment of flow-mediated dilation in humans: a methodological and physiological guideline.
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In neonatal patients with congenital diaphragmatic hernia, X-rays showed blurred lung on the hernia side. 45% of neonatal patients having congenital diaphragmatic hernia suffered from pulmonary arterial hypertension while the rate was 3% in postnatal patients. Recurrent pulmonary infection most happe ns in postnatal congenital diaphragmatic hernia patients..
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In a study examining right heart ventricular function, signi fi cant changes were found in the DGC of the right ventricle of rats administered monocrotaline (MCT), an alkaloid commonly used to induce pulmonary arterial hypertension, during development of right ventricular failure [161]. In a study by Yoshida et al. (C) Left ventricular assist device (LVAD) provides mechanical unloading of the heart. Proteins of the DGC and the cytoskeleton remodel in hearts. A study by Kyoi et al.
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Pulmonary arterial hypertension (PAH) can de- velop early in the patient’ s life, and the left-to-right shunt. Declining oxygen content in the systemic circula- tion can also develop into Eisenmenger syndrome, thereby increasing the severity of hypoxemia. Because the obstetricians believed that the patient was suffering from progressive dyspnea and might progress to acute cardiac insufficiency, it was necessary to operate to reduce her cardiac load and to reduce the probability of death.
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Role of diabetes mellitus and gastro- oesophageal reflux in the aetiology of idiopathic pulmonary fibrosis.. Pulmonary hypertension in patients with idiopathic pulmonary fibrosis. Significance of pulmonary arterial pressure and diffusion capacity of the lung as prognosticator in patients with idiopathic pulmonary fibrosis.. Mortality from pulmonary fibrosis increased in the United States from 1992 to 2003.
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Mild to moderate pulmonary hypertension in liver transplantation. Exercise intolerance in chronic thromboembolic pulmonary hypertension after pulmonary angioplasty. Differ- ences in ventilatory inefficiency between pulmonary arterial hyperten- sion and chronic thromboembolic pulmonary hypertension. Cardiopulmonary exercise testing to detect chronic thromboembolic pulmonary hyperten- sion in patients with normal echocardiography.
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Pulmonary hypertension (PH) is a lethal disease and defined as an increase in the mean pulmonary arterial pressure ≥ 25 mmHg at rest, as measured by right heart catheterization [1].. Hypoxia mediated pulmonary hypertension (HPH) belongs to group III PH according to the comprehensive clinical clas- sification of PH, normally accompanied by severe chronic obstructive pulmonary disease (COPD) and interstitial lung diseases [2].
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Nevertheless, every effort should be made to detect new onset severe TR and PHT in OLT waitlisted patients by performing regular TTE screening and having a high index of suspicion of TR and PHT at times of hepatic decompensation.. mPAP: Mean pulmonary arterial pressure. OLT: Orthotopic liver transplant. PAP: Pulmonary arterial pressure;. PHT: Pulmonary hypertension;. POPH: Porto-pulmonary hypertension. PVR: Pulmonary vascular resistance;. sPAP: Systolic pulmonary arterial pressure;.
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Effects of dexmedetomidine on porcine pulmonary artery vascular smooth muscle. The effects and mechanism of action of Dex on pulmonary arteries, however, have not been determined.. This study therefore aimed to investigate the effect of Dex on pulmonary arterial vascular smooth muscle by evaluating changes in isometric contraction tension. We then attempted to determine the effects of Dex on depolarization stimulation and receptor stimulation..
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Role of pulse pressure amplification in arterial hypertension: experts' opinion and review of the data. Accuracy of invasive arterial pressure monitoring in cardiovascular patients: an observational study. Reliability of effective arterial elastance using peripheral arterial pressure as surrogate for left ventricular end-systolic pressure
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NO should not be used in patients with severe left ven- tricular failure since the predominantly pulmonary arterial vaso- dilation (as opposed to pulmonary venodilation) could lead to pulmonary edema [78. To date, the benefi ts of inhaled NO in patients with ARDS are short - lived and mainly have shown a transient improvement in oxygenation without improving sur- vival.