Tìm thấy 20+ kết quả cho từ khóa "Skin Manifestations"
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Skin Manifestations of Internal Disease. Skin Manifestations of Internal Disease: Introduction. It is now a generally accepted concept in medicine that the skin can show signs of internal disease. Therefore, in textbooks of medicine one finds a chapter describing in detail the major systemic disorders that can be identified by cutaneous signs.
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Skin Manifestations of Internal Disease. a Also associated with systemic diseases.. b Reviewed in section on Purpura.. cReviewed in section on Papulonodular Skin Lesions.. d Favors plantar surface of the foot.. Purpuric lesions and livedo reticularis are found in association with painful ulcerations of the lower. These ulcers are often slow to heal, but when they do, irregularly shaped white scars are formed.
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The internal manifestations of Torre syndrome include multiple carcinomas of the gastrointestinal tract (primarily colon) as well as cancers of the larynx, genitourinary tract, and endometrium.
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Sontheimer RD: Skin manifestations of systemic autoimmune connective tissue disease: Diagnostics and therapeutics. Udey MC, Stanley JR: Pemphigus—diseases of antidesmosomal autoimmunity. JAMA PMID: 10450720]. JAMA PMID:
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Actual deposits of a particular drug or metal in the skin are seen with silver (argyria), where the skin appears blue-gray in color;. gold (chrysiasis), where the skin has a brown to blue-gray color. and clofazimine, where the skin appears reddish brown. The associated hyperpigmentation is accentuated in sun-exposed areas, and discoloration of the eye is seen with gold (sclerae) and clofazimine (conjunctivae).
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Complete examination of the skin and oral mucosa combined with a biopsy and direct immunofluorescence microscopy will aid in distinguishing these two entities. The peripheral active lesions in folliculitis decalvans are follicular pustules. Figurate Skin Lesions. (Table 54-6) In figurate eruptions, the lesions form rings and arcs that are usually erythematous but can be skin-colored to brown.
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Papulonodular Skin Lesions. (Table 54-15) In the papulonodular diseases, the lesions are elevated above the surface of the skin and may coalesce to form plaques. The location, consistency, and color of the lesions are the keys to their diagnosis. Table 54-15 Papulonodular Skin Lesions According to Color Groups
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Linear telangiectasias are seen on the face of patients with actinically damaged skin and acne rosacea, and they are found on the legs of patients with venous hypertension and essential telangiectasia. Patients with an unusual form of mastocytosis (telangiectasia macularis eruptiva perstans) and the carcinoid syndrome (see "Acne,". above) also have linear telangiectasias. Lastly, linear telangiectasias are found in areas of cutaneous inflammation. Actinically damaged skin.
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For example, about 95% of the patients with infectious mononucleosis who are given ampicillin will develop a rash.. petechiae of the palate. and desquamation of the involved skin, palms, and soles 5–20 days after onset of the eruption.
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Localized areas of decreased pigmentation are commonly seen as a result of cutaneous inflammation (Table 54-10) and have been observed in the skin overlying active lesions of sarcoidosis (see "Papulonodular Skin Lesions,".
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In lymphoma cutis there is a proliferation of malignant lymphocytes or histiocytes in the skin, and the clinical appearance resembles that of cutaneous lymphoid hyperplasia—infiltrated pink-red to red-purple papules and plaques.
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Resultant skin changes of the face, in particular telangiectasias, may mimic the clinical appearance of acne rosacea.. Acneiform eruptions (see "Acne,". above) and folliculitis represent the most common pustular dermatoses. An important consideration in the evaluation of follicular pustules is a determination of the associated pathogen, e.g., normal flora, Staphylococcus aureus, Pseudomonas aeruginosa ("hot tub".
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Patients with dermographism exhibit linear wheals following minor pressure or scratching of the skin. It is a common disorder, affecting ~5% of the population. Solar urticaria characteristically occurs within minutes of sun exposure and is a skin sign of one systemic disease—. In addition to the urticaria, these patients have subtle pitted scarring of the nose and hands. Cold urticaria is precipitated by exposure to the cold, and therefore exposed areas are usually affected.
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The arteritis leads to ischemia of the skin, and this explains the irregular outline of the purpura (see below).. Several types of infectious emboli can give rise to palpable purpura. These embolic lesions are usually irregular in outline as opposed to the lesions of LCV, which are circular in outline. The irregular outline is indicative of a cutaneous infarct, and the size corresponds to the area of skin that received its blood supply from that particular arteriole or artery.
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Poikiloderma does not imply a single disease entity—although becoming less common, it is seen in skin damaged by ionizing radiation as well as in patients with autoimmune connective tissue diseases, primarily dermatomyositis (DM), and rare genodermatoses (e.g., Kindler syndrome).. In scleroderma, the dilated blood vessels have a unique configuration and are known as mat telangiectasias.
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Capillary fragility leads to nonpalpable purpura in patients with systemic amyloidosis (see "Papulonodular Skin Lesions,". above), disorders of collagen production such as Ehlers-Danlos syndrome, and scurvy. In scurvy there are flattened corkscrew hairs with surrounding hemorrhage on the lower extremities, in addition to gingivitis. involved in the posttranslational modification of procollagen that is necessary for cross-link formation..
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Hypo melanotic areas—few to no. migration of melanoblasts from neural crest to ventral skin or failure of melanoblasts to survive or differentiate in these areas. Symm etric
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The differential diagnosis of PCT includes (1) porphyria variegata—the skin signs of PCT plus the systemic findings of acute intermittent porphyria. (2) drug-induced pseudoporphyria—the clinical and histologic findings are similar to PCT, but porphyrins are normal. (3) bullous dermatosis of hemodialysis—the same appearance as PCT, but porphyrins are usually normal or occasionally borderline elevated. (2) rubella—the eruption begins on the forehead and face and then spreads down the body
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Purple-colored papules and plaques are seen in vascular tumors, such as Kaposi's sarcoma (Chap. 182) and angiosarcoma, and when there is extravasation of red blood cells into the skin in association with inflammation, as in palpable purpura (see "Purpura,".
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Additional findings are discussed in the section on neurofibromas (see "Papulonodular Skin Lesions,". In incontinentia pigmenti, dyskeratosis congenita, and bleomycin pigmentation, the areas of localized hyperpigmentation form a pattern—swirled in the first, reticulated in the second, and flagellate in the third. Fixed drug eruptions recur in the exact same location as circular areas of erythema that can become bullous and then resolve as brown macules.