Tìm thấy 15+ kết quả cho từ khóa "Myocardial injury"
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A total of 16 patients (19.3%) had a major adverse cardiovascular event, of which 11 patients (13.3%) had myocardial injury. The L- arginine/ADMA ratio was significantly decreased at 4 – 24 h after surgery in patients suffering myocardial injury.. Early postoperative disturbances in the nitric oxide bioavailability might add to the pathogenesis of myocardial injury.
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The major- ity of myocardial injury occurred in patients with Mayo levels III and IV thrombi and during the first day after surgery (Fig. d According to the 1987 version without age correction Non-myocardial injury. (n = 89) Myocardial injury. without myocardial injury, those with myocardial injury developed more AKI vs. Among patients admit- ted to the ICU, those with myocardial injury had more endotracheal intubation vs.
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Association of intra-operative hypotension with acute kidney injury, myocardial injury and mortality in non-cardiac surgery:. Relationship Between Intraoperative Hypotension and Acute Kidney Injury After Living Donor Liver Transplantation: A Retrospective Analysis. Preoperative Risk and the Association between Hypotension and Postoperative Acute Kidney Injury..
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Conclusions: Compared with the group PR, sevoflurane had no benefit in the myocardial protection for the elderly patients with CHD. Myocardial injury can lead to more perioperative com- plications and prolonged hospitalization, especially for the elderly patients with coronary heart disease (CHD) undergoing major surgery.
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Thus, in this sub-study of the fore mentioned ran- domized controlled trial, we tested the hypothesis that intraoperative goal-directed therapy with IV colloid compared to crystalloid solutions will lead to less. myocardial injury.
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There is limited work published in the literature look- ing specifically to myocardial injury as a marker of out- come at the thoracic surgical population submitted to lung resection. In our study, we collected troponin I from all patients subjected to lung resection, regardless of the preoperative cardiac risk classification, and tropo- nin I levels were analyzed from the limit of detection since we were looking for myocardial injury and not just AMI.
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Association of Intraoperative Hypotension with acute kidney injury after noncardiac. https://doi.org . Impact of intraoperative hypotension on hospital stay in major abdominal surgery. https://doi.org/10.1097/ALN.0b013e318190b6dc.. Association between intraoperative hypotension and myocardial injury after vascular surgery. org/10.1097/ALN .
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Effect of remote ischaemic preconditioning on myocardial injury in patients undergoing coronary artery bypass graft surgery: a randomised controlled trial. Zarbock A, Schmidt C, Van Aken H, Wempe C, Martens S, Zahn PK, et al.. Effect of remote ischemic preconditioning on kidney injury among high-risk patients undergoing cardiac surgery. Ali ZA, Callaghan CJ, Lim E, Ali AA, Nouraei SAR, Akthar AM, et al.
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Preoperative heart rate and myocardial injury after non-cardiac surgery: results of a predefined secondary analysis of the VISION study. Belcher AW, Khanna AK, Leung S, et al. Berwanger O, Le Manach Y, Suzumura EA, et al. Gorka J, Polok K, Iwaniec T, et al. Khanna AK, Sessler DI, Sun Z, et al. Roshanov PS, Eikelboom JW, Crowther M, et al. Roshanov PS, Walsh M, Devereaux PJ, et al. Vascular Events in Noncardiac Surgery Patients Cohort Evaluation Study I, Spence J, LeManach Y, et al. Licker et al
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Preoperative heart rate and myocardial injury after non-cardiac surgery:.
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Cardiac hypoperfusion and the application of a large number of vasoactive drugs in patients with sepsis both may cause myocardial injury[17]. BNP on d3 (pg/ml) <. 681.5( n = 172) >. Septic shock 0.000. Table 4 Comparison of BNP and cTnI between the non-septic shock group and the septic shock group. Non-septic shock ( n = 139) Septic shock ( n = 94) P. BNP on d .
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Despite transient myocardial injury (elevated troponin I), no lethal arrhythmia or complications occurred perioperatively, and the patient recovered well and was discharged 1 week later..
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The cardiovascular complications of COVID-19 have been reported as myocardial injury, arrhythmias, myocarditis, and venous thromboembolism [18]. Xu et al. It is known that cardiac involvement and dysfunction are associated with a worse prognosis in COVID-19 patients [19]. The pathogenesis of cardiac involvement of COVID-19 might possibly be due to damage to the heart directly by the virus and the degeneration and necrosis of cells with hyaline thrombus formation in small vessels [20].
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Although a role for HtrA2/Omi has been suggested in response to myocardial injury or heart failure (Siu et al. XIAP is a fundamental conserved gene product among many species (Deveraux et al. Sanna et al. Chowdhury et al. ARC and FLIP are two endogenous apoptosis-suppressing proteins with high expression levels in muscle tissue (Irmler et al. Koseki et al. Abmayr et al. Heikaus et al. Yu et al.
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The cause of cardiac arrest could be bệnh nhân được hồi sinh tim phổi sống sót sau attributed to acute respiratory acidosis, acute khi xuất viện và sống sót sau khi myocardial injury (myocardial infarction or acute xuất viện với tình trạng thần kinh bình thường myocarditis) and the consequences of a cytokine hoặc suy giảm nhẹ.1 storm.
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Myocardial injury, ischemia- like electrocardiographic changes and arrhythmias, fre- quently occur in acute stroke patients, even in the ab- sence of primary heart disease [2]. Cardiac dysfunction. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material.
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Treatment of myocardial ischemia/reperfusion injury by ischemic and pharmacological Postconditioning. Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discovery. cardioprotective effects than limb remote ischemic postconditioning in rats with myocardial ischemia-reperfusion injury. The inflammatory response as a target to reduce myocardial ischaemia and reperfusion injury.
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Effect of remote ischemic preconditioning on kidney injury among high-risk patients undergoing cardiac surgery: a randomized clinical trial. Across-species transfer of protection by remote ischemic preconditioning with species-specific myocardial signal transduction by reperfusion injury salvage kinase and survival activating factor enhancement pathways.
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The study showed that treatment of Hes decreased the down-regulated PPARγ and Bcl2 apoptosis regulator expressions in myocardial infarcted diabetic hearts [17]. Short-term pretreatment with Hes protected against myocardial IR injury by suppressing myocardial apoptosis, the inflammatory response and oxidative stress via phosphoinositide 3-kinases/protein kinase B pathway activation and high mobility group box 1 protein inhibition [19].
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The largest changes in female hearts involved genes that regulate the circadian clock ( Dbp, Per3, Per2, BMal1 , and Npas2 ) which are known to impact myocardial ischemic injury. All changes in gene expression identified at day 11 returned to baseline by day 30.. These studies suggest that methamphetamine (dur- ing either the prenatal period or during early adulthood) may induce changes in gene expression that are sex- dependent and that persist after methamphetamine use has been discontinued..