Tìm thấy 20+ kết quả cho từ khóa "Stroke volume"
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In the Doppler group, volume optimization was guided by the stroke volume changes in accordance with pub- lished protocols [14, 15]. Fluid bo- luses were repeated until the stroke volume no longer increased by 10%. The 10% cut-off value is commonly used in studies involving the esophageal Doppler, and is based on measurement characteristics of the device [16].. During surgery, additional optimizations were under- taken in the same way whenever the stroke volume had decreased by ≥10%..
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Abbreviations: ROC Receiver operating characteristics, SV Stroke volume, ΔSV LRM Decrease in stroke volume by lung recruitment maneuver, SVV prone Stroke volume variation at time point T3, PPV prone Pulse pressure variation at time point T3, SVV supine Stroke volume variation at time point T0, PPV spine Pulse pressure variation at time point T0, AUC Area under the curve. ΔSV LRM. into account, Biais et al. [14] applied LRM for the assess- ment of fluid responsiveness.
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Assessment of stroke volume variation for prediction of fluid responsiveness using the modified FloTrac and PiCCOplus system. Effects of concentration and volume of 2-chloroprocaine on epidural anesthesia in volunteers. Accuracy of stroke volume variation in predicting fluid responsiveness: a systematic review and meta-analysis.. Stroke volume variation as a predictor of fluid responsiveness in patients undergoing one-lung ventilation.
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Left ventricular-arterial coupling as a predictor of stroke volume response to norepinephrine in septic shock – a. Background: Left ventricular-arterial coupling (VAC), defined as the ratio of arterial elastance (Ea) to left ventricular end-systolic elastance (Ees), is a key determinant of cardiovascular performance. This study aims to evaluate whether left VAC can predict stroke volume (SV) response to norepinephrine (NE) in septic shock patients..
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PPV 6 , pulse pressure variation during tidal volume at 6 ml/kg predicted body weight (PBW). PPV 8 , pulse pressure variation during tidal volume at 8 ml/kg PBW. Δ PPV 6–8 , change in value of pulse pressure variation after tidal volume challenge. SVV 6 , stroke volume variation during tidal volume at 6 ml/kg predicted body weight (PBW). SVV 8 , stroke volume variation during tidal volume at 8 ml/kg PBW. 8 , change in value of stroke volume variation after tidal volume challenge.
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SV, stroke volume. SVV, stroke volume variation. VTI-LVOT: velocity time integral of left ventricular outflow tract. 1 Correlation between the Left ventricular end-diastolic volume variation (LVEDVV) estimated by left ventricular short diameter of axle using TEE and stroke volume variation (SVV) obtained with the FloTrac/Vigileo monitor. reliability of cardiac output measurement obtained by this system was confirmed.
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MAP mean arterial pressure, HR heart rate, CO cardiac output, CI cardiac index, SV stroke volume, SVI stroke volume index, SVV stroke volume variation, VCV volume controlled ventilation, PCV-VG, pressure controlled ventilation with volume guaranteed. It is known that the CI decreases when a patient moves in the prone position. They found that decreases in CI in the prone position resulted from a decreased SV..
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Fluid challenges consisted with 250 ml of either bicarbonate Ringer solution, 6% hydroxyethyl starch or 5% albumin were provided to maintain optimal stroke volume index.. Hematocrit derived-plasma volume and colloid osmotic pressure was determined immediately before and 30 min after the fluid challenge. 30 min after the fluid challenge with 6% hydroxyethyl starch and 5%.
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Based on the assumption of α = 0.05, power = 0.8, a normal distribution and a standard deviation of stroke volume = 20% as well as clinical relevant different changes of stroke volume = 15% induced by fluid loading, a sample size of 30 was calculated (including two patients as potential drop-outs). Prediction of fluid respon- siveness was assessed by calculating the area under the receiver operating characteristic (ROC) curve for a stroke volume increase >.
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In this manner, the increased stroke volume may be a direct result of “ normalized ” vascular fi lling in the setting of systemic after- load reduction. These data support the conclusion of Morton and co - workers [68] that early stroke volume increases are caused by a “ shift to the right ” of the left ventricular pressure – volume curve (Frank – Starling mechanism)..
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The intraoperative cardiac and stroke volume index, central venous pressure, stroke volume variation, and systemic vascular resistance for patients in the GDFT group are presented in Fig.
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The predictability of dynamic preload indices depends on the volume of fluid challenge: a prospective observational study in the operating theater. Predictability of the respiratory variation of stroke volume varies according to the definition of fluid responsiveness. Stroke volume changes induced by a recruitment maneuver predict fluid responsiveness in patients with protective ventilation in the operating theater. Limitations of the Frank-Starling Mechanism.
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SV: Stroke Volume (60-100 mL) SVI: Stroke Volume Index (35-45 mL/m 2 ) CO: Cardiac Output (4.0-8.0 L/min) CI: Cardiac Output Index (2.6-3.8 L/min.m 2. sử dụng các pp đo cung lượng tim để điều. Mục tiêu Cardiac Index 3,3 - 6,0 l/ph/m2 Sử dụng SVRI (Systemic Vascular. SỬ DỤNG MOSTCARE THEO DÕI HUYẾT ĐỘNG LIÊN TỤC Ở BN SỐC NHIỄM TRÙNG. SỬ DỤNG MOSTCARE THEO DÕI HUYẾT ĐỘNG LIÊN TỤC Ở BN TAY CHÂN MIỆNG. SỬ DỤNG MOSTCARE THEO DÕI HUYẾT ĐỘNG LIÊN TỤC Ở BN SỐC SỐT XUẤT HUYẾT
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The fundamental cause of respiratory variation in per- ipheral arterial blood flow peak velocity was the respira- tory variation of stroke volume. From the results of the study, the △ Vpeak of carotid artery had more value than brachial artery in predicting fluid responsiveness. The reason may be that carotid artery had the advantage of anatomical location (closer to the heart). Thus, the blood flow of carotid artery is more sensitive to the change of stroke volume than brachial artery.
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Patients of the GDT group expe- rienced an individually tailored preloading with crystalloid fluids according to stroke volume optimization without risking volume overload. Apart from that, acute in- crease of pulmonary vascular resistance as part of BCIS might lead to distension of the right ventricle with a con- comitant decrease of coronary blood flow. Al- though cardiac and stroke volume index were significantly higher in the GDT group we were not able to reduce the risk for overall BCIS..
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In the IFM group (from April 2017) the radial line was connected to the fourth-generation Vigileo/Flotrac sys- tem (Edwards Lifesciences, Irvine, CA, USA) enabling continuous monitoring of stroke volume from pulse contour analysis. Once patients were in the prone position, we started to monitor stroke volume and a bolus of 3 ml/kg of Ringer’s lactate was administered over a 5 min period.
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0.0001), but global right ventricular ejection fraction (RVEF. p = 0.1607) and right ventricular stroke volume (RVSV. Conclusions: This data shows a preserved right ventricular ejection fraction and right ventricular stroke volume after anesthesia induction and initiation of positive pressure ventilation. However, the baso-apical right ventricular function is significantly reduced.
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USCOM Utrasonic Cardiac Output Monitoring 28 SVV Stroke Volume Variation 29 SVRI Systemic Vascular Resistance Index 30 ET % Ejection Time 31 FTc Flow Time Correct 32 INO Inotropic Index 7 33 MD Minute Distance 34 CPO Cardiac Power 35 PKN Potential & Kinetic Energy Ratio 36 SW Stroke Work 38 SVS Stroke Volume Saturation 39 Pmn Mean Pressure Gradient Across The Valve 40 GEDI Global End Diastolic Index 41 MAP Mean Arterial Pressure 42 APsys Arterial Pressure Systolic 43 APdia Arterial Pressure Diastolic
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However, in low-risk patients undergoing these surgical procedures optimizing stroke volume may have limited impact on outcome..
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Mean arterial pressure, heart rate, cardiac output (measured by electrical cardiometry), stroke volume, and systemic vascular resistance were recorded in three positions (supine, 15 0 , and 30 0 left lateral positions) before SAB, after SAB, and after delivery of the fetus.. Results: Before SAB, no significant hemodynamic changes were reported with left lateral tilting.