Tìm thấy 17+ kết quả cho từ khóa "Virulence gene"
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They observed that 3- oxo-C12-HSL inhibited agr QS expression and antagonized virulence gene expression in S.
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Bac- terial mutation rates and the selective advantages brought by the virulence gene to the bacteria lead to the complete inactivation of the integrated phage’s ability to form new particles and lyse the host, while maintaining the virulence gene functionality [43]..
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However, the gene-id based detection of genes from the KT strain was unfeasible due to incongruences between gene-ids from the KT strain and the ones deposited in the NCBI gene database, thus the 4-letter gene name was used to retrieve the genes of interest, resulting in a partial set of 125 genes for this analysis. Most of the known virulence genes present in this set were found in the sequences of the five poultry strains (Fig.
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Five antibiotic resistance genes and one virulence gene, CU052_28670, involved in immune evasion were detected on plasmid p345–185. harveyi 345 was deposited in GenBank with accession numbers CP025537 (ChI), CP025538 (ChII), CP025539 (plasmid p345–185) and CP025540 (plasmid p345–67).. harveyi 345 was isolated and studied because of its multidrug resistance and serious virulence to E.
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The deficiency of T3SS gene expression in the (p) ppGpp 0 mutant indicated that (p) ppGpp is required for T3SS expression in E. Down-regulation of the T3SS genes, accompanied by attenuated virulence and reduced growth, was also re- ported in the P.
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In support of our hypoth- esis, we found that not only specific degraded/absent virulence gene, but also certain allelic virulence protein sequences were linked to the intestinal or invasive life- style of the various Salmonella serovars or lineages.. The confirmation of previously described virulence levels associated with spe- cific allelic or absent virulence factors sup- ports the potential biological relevance of the new host- virulence factor allele associations detected in this study..
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Association of AMR genes, plasmid replicons, and virulence genes with different Salmonella serotypes using WGS. Serotypes were found to vary with regard to the pres- ence/absence of AMR coding gene, plasmid replicon, and virulence gene using WGS approach based on the odds ratio to evaluate their associations (Table 3). As highlighted previously, several major virulence genes were detected in all Salmonella isolates in our study (Additional file 2: Table S2).
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Identification of a virulence gene cluster of Mycobacterium tuberculosis by signature-tagged transposon mutagenesis
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Analysis of orthologs revealed substantial heterogeneity in physiological and virulence-related genes between gastric and EHS genomes. Most Helicobacter spp. Phylogenetic classification of gastric and enterohepatic Helicobacter species. For example, Helicobacter spp. jejuni genomes, while 692 were shared between just gastric and EHS genomes.. 1 a) 16s rRNA gene and b) pan-genome phylogenetic trees both differentiated gastric and EHS genomes.
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Var genes encode major virulence factors involved in immune evasion and the maintenance of chronic infections. In the human parasite P. falciparum , var gene recombination and diversification appear to be promoted by G-quadruplex (G4) DNA motifs, which are strongly associated with var genes in P.
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A heatmap of the 11 genes with differential NSY SNP counts (P <. 11 genes had fewer NSY mu- tation sites in the high-virulence group and more NSY SNP sites in the low-virulence group.. Of the 5 identified key virulence related genes, 4 were in Contig 3 and 1 was in Contig 1, which was con- sistent with the virulence related areas detected in the Manhattan map (Table 4).. For instance, there were 3 SNP sites in the gene A05389, at contig3:1317647, contig3:1317841 and con- tig3:1319579.
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The primary transcriptome of the Escherichia coli O104:H4 pAA plasmid and novel insights into its virulence gene expression and regulation. Comparative virulence characterization of the Shiga toxin phage-cured Escherichia coli O104:H4 and enteroaggregative Escherichia coli
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Several studies have extensively focused on the incidence of virulence determinants in Enterococci species reporting that a remarkable low occurrence of virulence genetic determinants was detected in Enterococcus isolates from foods or used as starters (Eaton and Gasson, 2001), but also in those from water and soil (Abriouel et al., 2008).
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Regulation of Virulence Gene Expression. In both toxin-mediated and non-toxin-mediated diseases due to S. aureus, the expression of virulence determinants associated with infection depends on a series of regulatory genes [e.g., accessory gene regulator (agr) and staphylococcal accessory regulator (sar)] that coordinately control the expression of many virulence genes. Staphylococcal surface proteins are synthesized during the bacterial exponential growth phase in vitro.
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The next 13 rings moving towards the inner part of the figure display regions of sequence similarity detected by BLAST comparison between the DNA of the reference genome and the DNA of the 13 compared T. forsythia virulence are labeled in the plot. forsythia virulence genes, only bspA (encoding the bacterial surface-associated protein) was not conserved in the ancient and most contempor- ary genomes (Fig.
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Virulence and molecular characterization of experimental isolates of the stripe rust pathogen (Puccinia striiformis) indicate somatic recombination.. GC transitions in the barley Ant18 gene. Variation in the AvrSr35 gene determines Sr35 resistance against wheat stem rust race Ug99. Current status of the gene-for-gene concept. Cladosporium fulvum effectors: weapons in the arms race with tomato. A Puccinia striiformis f.
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We show the utility of the browser for the analysis of putative virulence genes from the pathogen and the response of the host plant to Pst infection. This illustrates the immense value of ana- lysing a broad set of RNA-Seq data to provide insight into gene expression regulation during host-pathogen interactions.. Generating RNA-Seq data and its incorporation into the rust expression browser.
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As gene duplication obviously increases the complexity of the gene sequence types in the core virulence-related genes, we should pay more attention to the roles gene duplica- tion played in virulence evolution. cholerae with respect to virulence-related gene evolution and virulence surveil- lance, indicating that strains with more virulence-related genes may have an evolutionary advantage..
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Four of the 30 modules, FGSG_04220, FGSG_07052, FGSG_08028 and FGSG_10384, were hub regulators of virulence. FGSG_08028, the top regulator for M06, M24 and M49, activated genes of the three modules during wheat plant infection. Notably, both posi- tive and negative regulators were found in the virulence TRNs. 5 Fusarium graminearum subnetworks involved in the regulation of key phenotypes: sexual reproduction (a), virulence (b), mycotoxin production (c).
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Beauveria bassiana ERL836 and JEF-007 with similar virulence show different gene expression when interacting with cuticles of western flower thrips, Frankniella. But, in transcription level, ERL836 appeared to infect thrips easily, while JEF-007 appeared to have more difficulty. In the GO analysis of ERL836 DEGs (differentially expressed genes), the number of up-regulated genes was much larger than that of down-regulated genes, when compared to JEF-007 DEGs (more genes down-regulated).