Tìm thấy 13+ kết quả cho từ khóa "Oral intake"
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Level of ERAS understanding affects. practitioners’ practice and perception of early postoperative resumption of oral intake:. Background: Early postoperative resumption of oral intake is supposed to be safe and beneficial to patients recov- ery. However, practitioners still have great confusion and disagreement about postoperative resumption of oral intake..
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We speculate from these findings that the oral intake of the 12.5% carbohydrate nutritional solution can play a role in alleviating the hunger feeling of the participants to a certain degree, and the stomach could be emptied enough after the oral administration before 2 h in our volunteers..
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Further assessment of oral hydration should be continued after returning to the ward.. Postoperative oral intake of fluids is very important for children following general anesthesia. This prospective randomized trial provided evidence that administering oral hydration at early post-anesthesia time points was safe in those children, improving satisfaction and decreasing thirst. Perioperative oral intake of fluids is very important for children follow- ing general anesthesia..
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There are significant differences in the oral pain score between baseline and 30, 60, and 120 min after application. comments contained suggestions for improvements of the Episil ® device.. The results of this study showed that Episil ® is an effec- tive device to relieve oral pain in Japanese patients with moderate to severe treatment-related OM, and its dura- tion of action was determined to be long enough to sup- port the patients’ oral intake.
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Th e strong hallucinogenic activity of LSD was confi rmed by Albert Hofmann who did synthesize the compound. the pharmaco- logical activity can be obtained by oral administration of as small as 20–75 µ g of LSD. Th e ab- sorption of LSD from the digestive tract is rapid and distinct visual hallucination takes place 45 min–1 h aft er oral intake of about 20 µ g of LSD [1]. Assessment of the method. Th e R f values of LSD were 0.29 with the (A) solvent system and 0.55 with the (B) system.
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Due to a nearly complete pre- systemic elimination, it is poorly suited for oral administration. how- ever, it is mainly administered orally in order to achieve a prolonged effect.. Taken orally, it is ab- sorbed and is not subject to first-pass elimination.. Af- ter oral intake, it is slowly converted into an active metabolite. It is administered by infusion to achieve controlled hypotension under continuous close monitoring. Relaxation Smooth muscle cell.
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Written HIPAA consent has been obtained from the patient and is available for review.. instructed along with minimal oral intake in the preced- ing days due to her persistent nausea.. The anesthesia team was contacted by the patient’s nurse in the post anesthesia care unit (PACU) for persistent postoperative nausea, a frequent issue encountered in the PACU. The patient’s physical exam was unremarkable with normal vital signs.
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Figures 3.1 and 3.2) are being used for hair dyes. p-Phenylenediamine is rapidly absorbed into blood through mucous membranes of the digestive tract aft er its oral intake, and metabolized into quinonediimine, which acts as a cyto- toxin. As its poisoning symptoms aft er oral intake, vomiting, epigastralgia, edemas of the face, neck and pharynx, dyspnea, acute renal failure, rhabdomyolysis, hemolysis, methemoglo- binemia, hepatic disorders and others can occur [1, 2].
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A patient with a severe SRI requires early feeding within the first several days of care because the condition is likely to produce inadequate spontaneous intake over. the next 7 days. A moderate SRI, as commonly seen during a postoperative period without oral intake that exceeds 5 days, benefits from adequate feeding by day 5–7 if the patient was initially well nourished.
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Oxazepam: therapeutic, 0.5–2 µ g/mL. toxic, 0.2 µ g/mL (a single oral intake). fatal, 5.2–9 µ g/mL.. She had no history of smok- ing, and had quitted drinking alcohol since the appearance of the epileptic attack. Th ere were erythematous exanthemas and the characteristic stiff ness of the neck. All drugs for oral intake except phenytoin were discontinued on day 3 aft er admission. the functions of the kidney, liver and heart were aggravated. She died 12 days aft er admission.
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Its toxicity is observed aft er oral, percutaneous, intraperitoneal, intravenous and subcutaneous administrations. Aft er inhalation of hydrazoic acid vapor and oral intake of azide, respiratory stimulation and tachycardia appear, followed by metabolic acidosis, hypotension, respiratory suppression, bradycardia, convulsion and fi nally death. Metabolism and excretion of azide in humans have not been well studied [4].
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Time courses of plasma concentrations of cresol isomers and their conjugates in a cresol- poisoned patient after its oral intake.. Cresol poisoning cases. Th e blood concentrations of unconjugated cresol in fatal poisoning cases are 71–190 µ g/mL.. In the survived Cases 7–14, the blood specimens were sampled at the fi rst medical exami- nation. the plasma concentrations of unconjugated cresol were 9.5–58 µ g/mL..
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The time to oral intake was shorter in the LM group than in the C group (P <. The incidence of PONV and consumption of antiemetic agents were significantly lower in the LM group (P <. 0.05) than in the C group but not in the L group (P >. The satisfaction score of the LM group was significantly higher than that of the C and L groups (P <.
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The role of oral fluid intake following adeno-tonsillectomy. Feasibility of a small amount of water intake at early stage after general anesthesia in children
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Diseases of the Oral Mucosa. Table 32-1 Vesicular, Bullous, or Ulcerative Lesions of the Oral Mucosa. Lip and oral mucosa (buccal, gingival, lingual mucosa). Lasts about 1 week, but condition may be prolonged if secondarily infected.. Gingiva and oral mucosa. may be. accompanied by small vesicles on oral mucosa that rupture to form shallow ulcers;. Oral mucosa Fatigue, sore throat, malaise, fever, and. spontaneously in 2–4 weeks.
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POMC is an important gene in this pathway, which plays an important role in the regulation of feed intake. Furthermore, we found that some path- ways such as adipocytokine signaling pathway, insulin signaling pathway and melanogenesis pathway are involved in the energy homeostasis and regulation of feed intake in chicks, and genes including SOCS1, NFKB1, NFKBIA and PIK3R5 are also related to feed intake regulation..
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Residual feed intake Poultry. Calculation of the residual feed intake. The feed cost accounts for about 60 – 70% of the gross costs of poultry production (Fathi et al., 2019. In general, the residual feed intake (RFI) and feed conver- sion ratio (FCR) are used to determine feed ef fi ciency (Yuan et al., 2015).
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Significant risk factors for oral cancer in Western countries include sun exposure (lower lip) and tobacco and alcohol use. In India and some other Asian countries, smokeless tobacco mixed with betel nut, slaked lime, and spices is a common cause of oral cancer. Less common etiologies include syphilis and Plummer-Vinson syndrome (iron deficiency)..
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The goats on CV and CV + CC had higher total DM and ME intake than those fed GG and GG + CV (P <. The goats on the CV diet had the highest CP intake (P <. The highest ADF intake was observed in the CC diet and followed by CC + CV, GG + CC, and GG diets, while NDF intake was not signi fi cantly different among the dietary treatments.
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Usually lateral tongue, rarely elsewhere on oral mucosa. found in HIV carriers in all risk groups for AIDS. Due to EBV. responds to. Anywher e on skin and oral mucosa.